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celldamage 450 px

Rutgers scientists have discovered that a protein (p62) which is supposed to prevent cell damage is not working efficiently in laboratory mice with heart and liver disease. Wei-Xing-Zong, a professor in the Department of Chemical Biology in the Ernest Mario School of Pharmacy and leader of the study, said the damage occurred because another protein, (TRIM21) – which should activate the body’s response system to fight off bacteria and virus – did the opposite in these seriously ill mice and shut the antioxidant protein down, preventing it from doing its job.

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